First, my best wishes as we acknowledge the passing of what I hope was a happy and healthy year for you!
So yes, another year… and we take a second to reflect on many things, including the march forward of ‘Father Time’.
That ‘March’ won’t stop, but there is plenty we have learned about ‘Managing-the-March’ and optimizing our aging process.
One thing we know is the loss of healthy adult stem cell function can accelerate as time passes, so we must develop a key line of defense and proactively support our stem cell functionality.1-3
Our stem cell system is extremely elegant, and like any complex system, the probability of a problem increases as a function of time… it’s natural.
What’s critical now, is to do everything possible to support this ecosystem in such a way that our stem cells continue to replicate in as healthy and consistent a manner as possible, regardless of the passage of time.
We also know what is pivotal to healthy stem cell function are healthy telomeres. In fact, this discovery is so important that the research team was rewarded a Nobel Prize in 2009.
So, the point is that with proper support, stem cells, unlike any other cells in the body, have a strong likelihood to self-renew5 supporting the healthy adult body's ability to self-heal and retain its youthful vigor.
The key takeaway is:
To support your Stem Cells, you need to also support your Telomeres.
The following are just a few of the external benefits associated with supporting your stem cell system through longer telomeres:
- A reduction in age spots and fine lines
- A reduced rate of aging for hair, skin and nails
- Lower probability of diabetes – the telomeres of diabetics and pre-diabetics are shorter
- Increased likelihood of losing unwanted pounds and maintaining your target weight. As people gained weight, the length of telomeres was found to be shorter
- Lower probability of osteoporosis - short telomeres are correlated with aging bone and the loss of bone mineral density
- Lower probability of chronic headaches - short telomeres are also associated with migraines in women
Working from this key premise, Age Reversal Inc. designed TeloSC, a nutraceutical formulated to support both your STEM CELL and TELOMERE functions.
It is designed and priced to be as accessible and affordable for as many people as possible.
If you make resolution at this time of year, I suggest it is to add TeloSC to your health regimen.
One other point.
As the year winds down… there is plenty to appreciate, reflect upon and sometimes stress about. There is no stress with TeloSC, as my guarantee is all about ‘No-Stress-Is-Best’!
“No-Stress-Is-Best” Money-Back Guarantee. Take all the time you need – stress-free – to see and feel the positive results that our remarkable products give you. So, NO STRESS. If you are not happy with your results, please notify us up to 365 days after your original purchase by calling 800.881.5346 and you will receive a 100% refund (less shipping, if applicable). This is straightforward and simple – no questions asked and no fine print to stress about.
Have a great Holiday, and as always – ‘More Life!’
1. Telomeres are the protective caps on the ends of the strands of DNA called chromosomes, which house our genomes. In young humans, telomeres are about 8,000-10,000 nucleotides long. They shorten with each cell division, however, and when they reach a critical length the cell stops dividing or dies. Flowers A, Lee JY, Acosta S, et al. NT-020 treatment reduces inflammation and augments Nrf-2 and Wnt signaling in aged rats. J Neuroinflammation. 2015;12:174.
2. Bickford PC, Kaneko Y, Grimmig B, et al. Nutraceutical intervention reverses the negative effects of blood from aged rats on stem cells. Age (Dordr). 2015;37(5):103.
3. Gu SG, Wang CJ, Zhao G, et al. Role of vitamin D in regulating the neural stem cells of mouse model with multiple sclerosis. Eur Rev Med Pharmacol Sci. 2015;19(21):4004-11.
4. Goodell MA, Rando TA. Stem cells and healthy aging. Science. 2015;350(6265):1199-204.
5. Schultz MB, Sinclair DA. When stem cells grow old: phenotypes and mechanisms of stem cell aging. Development. 2016;143(1):3-14.