Healthy Life Extension
New Website Expands Happiness and Longevity Mastery
Dear Future Centenarian,
Twenty years ago, I thought my time would be best spent on œtechnologies that will deliver radical life extension breakthroughs to the exclusion of supplements and some other pursuits.
Then two funny things happened.
We started to understand we have more control over our physical and mental destinies than we ever thought possible.
And recently, new technologies started uncovering age-related gene-expressing compounds. As it turns out, many of these compounds are natural herbs, vitamins and more.
I wasn™t the only one to realize the latter of course. Drug companies did too.
So far, they haven™t been able to regulate fish oil for example, but the FDA has approved a prescription drug formulation of fish oils called Lovaza from GlaxoSmithKlein. And it generates about $1 billion in annual sales!!!
If you buy Lovaza from an online pharmacy, the cheapest I™ve seen it is $139.20 per month. That™s 5.3 times as much as what I consider to be a BETTER formula from Life Extension.
(If you™re not a Life Extension member, email me for important info.)
So here are the points:
- Genes are an important part of the picture¦ but you are in control of the rest.
- Many supplements work on your genes in a positive way.
- If pharmaceutical companies sell supplements as drugs, they need to test them for years in order to get approval, while the private sector can sell them affordably as nutritional supplements as long as they are proven safe.
Just because supplements don™t need drug approval doesn™t mean they get developed overnight. My favorite, StemCode, has over 30 years of research for the underlying science behind it¦ and the validating technology was in the works for over a decade.
StemCode has more supporting data than I can even start to describe here, but it™s all at www.YourMaxLifeSolutions.com. As many as five more breakthrough supplements may be released over the next 18 months or so.
These, once validation is complete, are:
- Third generation Telomere formula
- Aging Brain formula which works on an entire network of aging brain and Alzheimer™s genes
- Anti-Aging formula which stimulates a whole network of longevity genes
- Anti-Inflammatory formula which modulates an entire network of genes that contribute to inflammation
- Anti-Obesity formula which helps control a full network of genes that make you fat
All will be available first from MaxLife Solutions.
There™s much more to the site though. You™ll not only see how to improve your health and extend your longevity, but you get to master the secrets of overcoming mental blocks that keep you from integrating the life enhancing habits that lead you to maximum Health, Happiness and Longevity.
This site can literally change your world.. It took a year to build, and you can now get some amazingly valuable free bonuses worth thousands of dollars during the launch period.
I urge you to visit now. www.YourMaxLifeSolutions.com
LATEST HEADLINES FROM FIGHT AGING!
SENS RESEARCH FOUNDATION'S AGE-BREAKER RESEARCH PROGRAMS Friday, MarchÂ 8, 2013
One of the root causes of aging is the formation of advanced glycation end-products (AGEs), something that happens much faster in a diabetic metabolism, but which nonetheless happens to all of us and causes progressively greater harm as the years pass.
AGEs gum together and disable vital protein machinery, and also hammer on cell receptors in ways that cause chronic inflammation and other ills.
INVESTIGATING THE MECHANISMS OF LIVER REGROWTH Friday, MarchÂ 8, 2013
The liver is one of the few organs capable of significant regeneration in humans - but even this is more a case of compensatory growth than true regeneration of the sort seen in lower animals.
Still, there is probably value in finding out how and why this happens in the liver and not in other organs: "[Researchers have] identified a protein complex that acts as a molecular switch turning on a self-regeneration program in the liver.
SUGGESTING THAT CALORIE RESTRICTION PRIMARILY OPERATES ON MITOCHONDRIAL FUNCTION Thursday, MarchÂ 7, 2013
Mitochondrial damage is important in aging, and a range of evidence suggests it to be perhaps the most important contribution to aging.
You might look at the membrane pacemaker theory of aging for example, which points to differences in susceptibility to mitochondrial damage between similar species with divergent life spans, where greater damage resistance correlates to longer life spans.
A SWITCH TO INCREASE PLASTICITY IN THE ADULT BRAIN Thursday, MarchÂ 7, 2013
Given an easy switch to increase the plasticity of the adult brain, boosting the pace at which new neurons and new neural connections are formed, researchers will gather much more data in the years ahead as to how effective this might be as a stop-gap therapy to slow or compensate for some of the effects of aging:
SEXUAL ACTIVITY AND NEUROGENESIS RATES IN MICE Wednesday, MarchÂ 6, 2013
With hindsight, it seems that this should be a fairly obvious development.
Given that evolution leads to organisms that adjust the operation of their metabolism in response to the prospects for food availability (see calorie restriction and related mechanisms), since that impacts reproductive success and thus evolutionary fitness, then it shouldn't be surprising to find that these organisms also do so based on the prospects for actual reproductive activity.
MORE VISCERAL FAT MEANS A GREATER RISK OF INTESTINAL CANCER Wednesday, MarchÂ 6, 2013
We know that removing visceral fat extends life in mice - even a drastic measure such as surgery to remove the fat increases mouse life span.
Given that mice are little cancer factories, it shouldn't be surprising to see that at least part of this effect on life expectancy stems from reduced incidence of cancer:
A "CALCIUM HYPOTHESIS" OF DECLINING VISION WITH AGE Tuesday, MarchÂ 5, 2013
Researchers here propose that a mechanism associated with age-related cognitive decline is also involved in the poorly understood general declines in vision that occur with age.
From the perspective of SENS and aging as damage, this is exactly the sort of thing we'd expect to be a secondary consequence of one of the fundamental changes that drive aging, such as a build-up of aggregates or damage to cell mitochondria.
ON METHIONINE RESTRICTION Tuesday, MarchÂ 5, 2013
Methionine is an essential amino acid, one that we do not manufacture ourselves but must obtain from what we eat.
It seems that a large fraction of the benefits of calorie restriction derive from alterations in metabolism that are based on sensing levels of methionine. Here is a review:
A REVIEW OF KNOWN LINKS BETWEEN GROWTH HORMONE AND AGING Monday, MarchÂ 4, 2013
The longest-lived genetically engineered mice are those in which growth hormone or growth hormone receptors have been diminished or removed entirely.
This review looks at some of what is known about the mechanisms involved in this extended life: "Studies in mutant, gene knock-out and transgenic mice have demonstrated that growth hormone
(GH) signaling has a major impact on aging and longevity.
CASTING DOUBT ON LATENT REGENERATIVE MECHANISMS IN MAMMALS Monday, MarchÂ 4, 2013
Demonstrations such as the unusual regenerative capacity of MRL mice have bolstered the idea that we mammals retain the vestiges of an ability to regenerate shared with lower animals such as salamanders - but suppressed or buried in some way.
Hence work on deciphering the mechanisms of limb and organ regeneration in a variety of species could lead to the ability to turn on similar regeneration in humans.